The following are the all of the articles that have been tagged as and being related to Slow Poison that can be found here at Musings From Us, for your enjoyment.
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In an earlier post, I mentioned some reasons to avoid altered corn sweeteners. In the next few days, I’ll tell you exactly what this slow poison has taken away from our bodies and why these things are so important to make our bodies function properly.
Today’s focus is on the hormone, Leptin. According to Wikipedia:
Leptin acts on receptors in the hypothalamus of the brain where it inhibits appetite by (1) counteracting the effects of neuropeptide Y (a potent feeding stimulant secreted by cells in the gut and in the hypothalamus); (2) counteracting the effects of anandamide (another potent feeding stimulant that binds to the same receptors as THC, the primary active ingredient of marijuana); and (3) promoting the synthesis of ?-MSH, an appetite suppressant. This inhibition is long-term, in contrast to the rapid inhibition of eating by cholecystokinin (CCK) and the slower suppression of hunger between meals mediated by PYY3-36. The absence of a leptin (or its receptor) leads to uncontrolled food intake and resulting obesity. Several studies have shown that fasting or following a very-low-calorie diet (VLCD) lowers leptin levels. It might be that on short-term leptin is an indicator of energy balance. This system is more sensitive to starvation than to overfeeding. That is, leptin levels do not rise extensively after overfeeding. It might be that the dynamics of leptin due to an acute change in energy balance are related to appetite and eventually to food intake. Although this is a new hypothesis, there are already some data that support it.